Color Atlas of Pathology: Pathologic Principles, Associated by Ursus-Nikolaus Riede, Martin Werner

By Ursus-Nikolaus Riede, Martin Werner

The pocket atlas is a superb reference paintings that offers pathologic findings in all clinically correct fields, utilizing top quality photos and concise, good dependent explanatory texts. pictures comprise colour photographs and micrographs. Electron microscopic pictures are incorporated the place worthy. transparent schematic drawings illustrate approaches akin to the process infections, and so on. textual content bins on scientific and morphological points, on problems, and with easy-to-remember mnemonic summaries are trigger in colour.

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Extra info for Color Atlas of Pathology: Pathologic Principles, Associated Diseases, Sequelae

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Actin filaments are 6 nm thick and usually lie beneath the cell membrane. They function as a sort of intracellular musculature. They attach to the same membrane molecules as do the fibronectin filaments outside. Microtubular Lesions Polymerization Block Pathogenesis: Inhibited tubulin self-aggregation inhibits these processes: — Mitosis; — Discharge of secretions; — Release of lysosomes. Example: Colchicine therapy for acute gout (p. 102). Microfilament Lesions Paired Helical Filaments Definition: These lesions appear as thickened coarse filaments resembling braids ( A2) in nerve cells.

108). This in turn causes liver cirrhosis (p. 42) and glomerular microcysts in the kidney. Clinical presentation: Autosomal recessive hereditary disease involving cerebral retardation with epilepsy, muscular hypotonia, spastic paralysis, deafness, and hepatic insufficiency. The disorder is rare and leads to death in infancy. Adrenoleukodystrophy Synonym: Familial Addison’s disease. Definition: Hereditary neural and metabolic disease. Pathogenesis: A defective peroxisomal assembly factor (PEX-gene) results in a peroxisomal enzyme defect.

Disproportional membrane and enzyme increase is indicative of poor adaptation to a deficiency in essential building blocks, such as riboflavin. The clinical result is an increased quantity of membranes with few enzymes. This sustains a reduced level of cell function in the presence of an extreme deficiency. Riede / Werner, Color Atlas of Pathology © 2004 Thieme All rights reserved. Usage subject to terms and conditions of license. A Proliferation of mitochondrial cristae (TEM) x 20000 B Loss of mitochondrial cristae (TEM) x 10000 C Matrix inclusion (TEM) x 5000 D Crista inclusions (TEM) x 20000 E Crista inclusions (mitochondrial myopathy) (TEM) x 40000 Riede / Werner, Color Atlas of Pathology © 2004 Thieme All rights reserved.

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